Theory

  1. Isn't targeting inflammation just addressing a symptom? As long as the spike protein remains won't the inflammation continue? Or am I oversimplifying the entire pathophysiology...

  2. Can I ask how melatonin helps? I take it regularly but very small doses as it tends to knock me about the next day. I take 1.5mg at night and sleep better than without. I find my symptoms are a little better the next day.

  3. https://www.walshmedicalmedia.com/open-access/chlorpheniramine-maleate-nasal-spray-in-covid19-patients-case-series.pdf

  4. "The pathophysiology consists of any initiating event that causes an increase in acetylcholine release that will enhance depolarization at postjunctional membrane of muscle fibers. This causes a muscle contraction due to the increased release of calcium. As the same inciting event recurs, there is a continuous contracture of sarcomeres which forms a trigger point. It is also believed that the repetitive stimulation induces a sense of hypoxia within the muscle, which causes sensitization of nociceptors [4,5]. As evident by the traumatic nature of the disease, severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) has been found to have intermediate and long-term effects on fatigue, respiratory function and carditis. The pain-related symptoms including myalgia and arthalgias account for 36% in a recent study [6]. Along with these symptoms, it also has been shown to cause lower limb weakness due to possible sclerosis or hypoxia [7,8]."

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